Cargando…
M19 Modulates Skeletal Muscle Differentiation and Insulin Secretion in Pancreatic β-Cells through Modulation of Respiratory Chain Activity
Mitochondrial dysfunction due to nuclear or mitochondrial DNA alterations contributes to multiple diseases such as metabolic myopathies, neurodegenerative disorders, diabetes and cancer. Nevertheless, to date, only half of the estimated 1,500 mitochondrial proteins has been identified, and the funct...
Autores principales: | Cambier, Linda, Rassam, Patrice, Chabi, Béatrice, Mezghenna, Karima, Gross, René, Eveno, Eric, Auffray, Charles, Wrutniak-Cabello, Chantal, Lajoix, Anne-Dominique, Pomiès, Pascal |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3282743/ https://www.ncbi.nlm.nih.gov/pubmed/22363741 http://dx.doi.org/10.1371/journal.pone.0031815 |
Ejemplares similares
-
Regulation of Skeletal Muscle Oxidative Capacity and Muscle Mass by SIRT3
por: Lin, Ligen, et al.
Publicado: (2014) -
SIRT3, a Mitochondrial NAD(+)-Dependent Deacetylase, Is Involved in the Regulation of Myoblast Differentiation
por: Abdel Khalek, Waed, et al.
Publicado: (2014) -
Overexpression of the Mitochondrial T3 Receptor p43 Induces a Shift in Skeletal Muscle Fiber Types
por: Casas, François, et al.
Publicado: (2008) -
Mice Lacking the p43 Mitochondrial T3 Receptor Become Glucose Intolerant and Insulin Resistant during Aging
por: Bertrand, Christelle, et al.
Publicado: (2013) -
Correction: Mice Lacking the p43 Mitochondrial T3 Receptor Become Glucose Intolerant and Insulin Resistant during Aging
por: Bertrand, Christelle, et al.
Publicado: (2014)