Cargando…
JunB contributes to Id2 repression and the epithelial–mesenchymal transition in response to transforming growth factor–β
The process of epithelial–mesenchymal transition (EMT) in response to transforming growth factor–β (TGF-β) contributes to tissue fibrosis, wound healing, and cancer via a mechanism that is not fully understood. This study identifies a critical role of JunB in the EMT and profibrotic responses to TGF...
Autores principales: | Gervasi, Megan, Bianchi-Smiraglia, Anna, Cummings, Michael, Zheng, Qiao, Wang, Dan, Liu, Song, Bakin, Andrei V. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3307698/ https://www.ncbi.nlm.nih.gov/pubmed/22391036 http://dx.doi.org/10.1083/jcb.201109045 |
Ejemplares similares
-
Boning up on JunB
por: Dove, Alan W.
Publicado: (2004) -
Abnormally expressed JunB transactivated by IL-6/STAT3 signaling promotes uveal melanoma aggressiveness via epithelial–mesenchymal transition
por: Gong, Chaoju, et al.
Publicado: (2018) -
JunB Is Critical for Survival of T Helper Cells
por: Hsieh, Tsunghan, et al.
Publicado: (2022) -
JunB: a paradigm for Jun family in immune response and cancer
por: Ren, Fu-jia, et al.
Publicado: (2023) -
m6A RNA methylation regulates the transcription factors JUN and JUNB in TGF-β-induced epithelial–mesenchymal transition of lung cancer cells
por: Suphakhong, Kusuma, et al.
Publicado: (2022)