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Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing
BACKGROUND: A number of theories have proposed possible mechanisms that may explain the high rates of comorbidity between posttraumatic stress disorder (PTSD) and persistent pain; however, there has been limited research investigating these factors. OBJECTIVE: The present study sought to prospective...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Co-Action Publishing
2011
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3402105/ https://www.ncbi.nlm.nih.gov/pubmed/22893804 http://dx.doi.org/10.3402/ejpt.v2i0.5652 |
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author | Carty, Jessica O'Donnell, Meaghan Evans, Lynette Kazantzis, Nikolaos Creamer, Mark |
author_facet | Carty, Jessica O'Donnell, Meaghan Evans, Lynette Kazantzis, Nikolaos Creamer, Mark |
author_sort | Carty, Jessica |
collection | PubMed |
description | BACKGROUND: A number of theories have proposed possible mechanisms that may explain the high rates of comorbidity between posttraumatic stress disorder (PTSD) and persistent pain; however, there has been limited research investigating these factors. OBJECTIVE: The present study sought to prospectively examine whether catastrophizing predicted the development of PTSD symptoms and persistent pain following physical injury. DESIGN: Participants (N=208) completed measures of PTSD symptomatology, pain intensity and catastrophizing during hospitalization following severe injury, and 3 and 12 months postinjury. Cross-lagged path analysis explored the longitudinal relationship between these variables. RESULTS: Acute catastrophizing significantly predicted PTSD symptoms but not pain intensity 3 months postinjury. In turn, 3-month catastrophizing predicted pain intensity, but not PTSD symptoms 12 months postinjury. Indirect relations were also found between acute catastrophizing and 12-month PTSD symptoms and pain intensity. Relations were mediated via 3-month PTSD symptoms and 3-month catastrophizing, respectively. Acute symptoms did not predict 3-month catastrophizing and catastrophizing did not fully account for the relationship between PTSD symptoms and pain intensity. CONCLUSIONS: Findings partially support theories that propose a role for catastrophizing processes in understanding vulnerability to pain and posttrauma symptomatology and, thus, a possible mechanism for comorbidity between these conditions. |
format | Online Article Text |
id | pubmed-3402105 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2011 |
publisher | Co-Action Publishing |
record_format | MEDLINE/PubMed |
spelling | pubmed-34021052012-08-14 Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing Carty, Jessica O'Donnell, Meaghan Evans, Lynette Kazantzis, Nikolaos Creamer, Mark Eur J Psychotraumatol Basic Research Article BACKGROUND: A number of theories have proposed possible mechanisms that may explain the high rates of comorbidity between posttraumatic stress disorder (PTSD) and persistent pain; however, there has been limited research investigating these factors. OBJECTIVE: The present study sought to prospectively examine whether catastrophizing predicted the development of PTSD symptoms and persistent pain following physical injury. DESIGN: Participants (N=208) completed measures of PTSD symptomatology, pain intensity and catastrophizing during hospitalization following severe injury, and 3 and 12 months postinjury. Cross-lagged path analysis explored the longitudinal relationship between these variables. RESULTS: Acute catastrophizing significantly predicted PTSD symptoms but not pain intensity 3 months postinjury. In turn, 3-month catastrophizing predicted pain intensity, but not PTSD symptoms 12 months postinjury. Indirect relations were also found between acute catastrophizing and 12-month PTSD symptoms and pain intensity. Relations were mediated via 3-month PTSD symptoms and 3-month catastrophizing, respectively. Acute symptoms did not predict 3-month catastrophizing and catastrophizing did not fully account for the relationship between PTSD symptoms and pain intensity. CONCLUSIONS: Findings partially support theories that propose a role for catastrophizing processes in understanding vulnerability to pain and posttrauma symptomatology and, thus, a possible mechanism for comorbidity between these conditions. Co-Action Publishing 2011-04-29 /pmc/articles/PMC3402105/ /pubmed/22893804 http://dx.doi.org/10.3402/ejpt.v2i0.5652 Text en © 2011 Jessica Carty et al. http://creativecommons.org/licenses/by-nc/3.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution-Noncommercial 3.0 Unported License, permitting all non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Basic Research Article Carty, Jessica O'Donnell, Meaghan Evans, Lynette Kazantzis, Nikolaos Creamer, Mark Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title | Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title_full | Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title_fullStr | Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title_full_unstemmed | Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title_short | Predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
title_sort | predicting posttraumatic stress disorder symptoms and pain intensity following severe injury: the role of catastrophizing |
topic | Basic Research Article |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3402105/ https://www.ncbi.nlm.nih.gov/pubmed/22893804 http://dx.doi.org/10.3402/ejpt.v2i0.5652 |
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