Cargando…
Postconditioning with Inhaled Carbon Monoxide Counteracts Apoptosis and Neuroinflammation in the Ischemic Rat Retina
PURPOSE: Ischemia and reperfusion injury (I/R) of neuronal structures and organs is associated with increased morbidity and mortality due to neuronal cell death. We hypothesized that inhalation of carbon monoxide (CO) after I/R injury (‘postconditioning’) would protect retinal ganglion cells (RGC)....
Autores principales: | Schallner, Nils, Fuchs, Matthias, Schwer, Christian I., Loop, Torsten, Buerkle, Hartmut, Lagrèze, Wolf Alexander, van Oterendorp, Christian, Biermann, Julia, Goebel, Ulrich |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3460901/ https://www.ncbi.nlm.nih.gov/pubmed/23029526 http://dx.doi.org/10.1371/journal.pone.0046479 |
Ejemplares similares
-
Carbon Monoxide Abrogates Ischemic Insult to Neuronal Cells via the Soluble Guanylate Cyclase-cGMP Pathway
por: Schallner, Nils, et al.
Publicado: (2013) -
Correction: Carbon Monoxide Abrogates Ischemic Insult to Neuronal Cells via the Soluble Guanylate Cyclase-cGMP Pathway
por: Schallner, Nils, et al.
Publicado: (2013) -
Inhalative preconditioning with hydrogen sulfide attenuated apoptosis after retinal ischemia/reperfusion injury
por: Biermann, Julia, et al.
Publicado: (2011) -
Argon Inhalation Attenuates Retinal Apoptosis after Ischemia/Reperfusion Injury in a Time- and Dose-Dependent Manner in Rats
por: Ulbrich, Felix, et al.
Publicado: (2014) -
The Carbon monoxide releasing molecule ALF-186 mediates anti-inflammatory and neuroprotective effects via the soluble guanylate cyclase ß1 in rats’ retinal ganglion cells after ischemia and reperfusion injury
por: Ulbrich, Felix, et al.
Publicado: (2017)