Cargando…
Desipramine Pretreatment Improves Sympathetic Remodeling and Ventricular Fibrillation Threshold after Myocardial Ischemia
Abnormal increase in sympathetic nerve sprouting was responsible for the ventricular arrhythmogenesis after myocardial infarction. This study investigated whether the norepinephrine transporter inhibitor, desipramine, can modulate sympathetic remodeling and ventricular fibrillation threshold (VFT) a...
Autores principales: | Wu, Xiaolin, Jiang, Hong, Yu, Lilei, Hu, Xiaorong, Liu, Wenwei |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Hindawi Publishing Corporation
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3471026/ https://www.ncbi.nlm.nih.gov/pubmed/23093858 http://dx.doi.org/10.1155/2012/732909 |
Ejemplares similares
-
Cytoprotection of rat hepatocytes by desipramine in a model of simulated ischemia/reperfusion
por: Shin, Jun-Kyu, et al.
Publicado: (2021) -
Sympathetic Nervous System Mediates Cardiac Remodeling After Myocardial Infarction in a Circadian Disruption Model
por: Wang, Yuhong, et al.
Publicado: (2021) -
Anti-inflammatory properties of desipramine and fluoxetine
por: Roumestan, Caroline, et al.
Publicado: (2007) -
Renal Denervation Reduced Ventricular Arrhythmia After Myocardial Infarction by Inhibiting Sympathetic Activity and Remodeling
por: Zhang, Wen‐hui, et al.
Publicado: (2018) -
Effect of renal sympathetic denervation on ventricular and neural remodeling
por: Wang, L., et al.
Publicado: (2018)