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Necdin modulates proliferative cell survival of human cells in response to radiation-induced genotoxic stress
BACKGROUND: The finite replicative lifespan of cells, termed cellular senescence, has been proposed as a protective mechanism against the proliferation of oncogenically damaged cells, that fuel cancer. This concept is further supported by the induction of premature senescence, a process which is act...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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BioMed Central
2012
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3495902/ https://www.ncbi.nlm.nih.gov/pubmed/22691188 http://dx.doi.org/10.1186/1471-2407-12-234 |