Cargando…
Ubiquitin Carboxy-Terminal Hydrolase L1 (UCH-L1) is increased in cerebrospinal fluid and plasma of patients after epileptic seizure
BACKGROUND: Clinical and experimental studies have demonstrated that seizures can cause molecular and cellular responses resulting in neuronal damage. At present, there are no valid tests for assessing organic damage to the brain associated with seizure. The aim of this study was to investigate cere...
Autores principales: | Mondello, Stefania, Palmio, Johanna, Streeter, Jackson, Hayes, Ronald L, Peltola, Jukka, Jeromin, Andreas |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3500207/ https://www.ncbi.nlm.nih.gov/pubmed/22931063 http://dx.doi.org/10.1186/1471-2377-12-85 |
Ejemplares similares
-
Ubiquitin C-terminal hydrolase L1 (UCH-L1): structure, distribution and roles in brain function and dysfunction
por: Bishop, Paul, et al.
Publicado: (2016) -
Ubiquitin C-Terminal Hydrolase-L1 (UCH-L1) in Prediction of Computed Tomography Findings in Traumatic Brain Injury; a Meta-Analysis
por: Ramezani, Fatemeh, et al.
Publicado: (2018) -
Ubiquitin Carboxyl-Terminal Hydrolases (UCHs): Potential Mediators for Cancer and Neurodegeneration
por: Sharma, Amit, et al.
Publicado: (2020) -
Ubiquitin carboxy-terminal hydrolase L1 (UCHL1) S18Y polymorphism in Alzheimer's disease
por: Zetterberg, Madeleine, et al.
Publicado: (2010) -
Assessment of Serum UCH-L1 and GFAP in Acute Stroke Patients
por: Ren, Changhong, et al.
Publicado: (2016)