Cargando…
High glucose mediates endothelial-to-chondrocyte transition in human aortic endothelial cells
BACKGROUND: Vascular calcification is one of the common complications in diabetes mellitus. Many studies have shown that high glucose (HG) caused cardiovascular calcification, but its underlying mechanism is not fully understood. Recently, medial calcification has been most commonly described in the...
Autores principales: | Tang, Rining, Gao, Min, Wu, Min, Liu, Hong, Zhang, Xiaoliang, Liu, Bicheng |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2012
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3502155/ https://www.ncbi.nlm.nih.gov/pubmed/22998723 http://dx.doi.org/10.1186/1475-2840-11-113 |
Ejemplares similares
-
Angiotensin II mediates the high-glucose-induced endothelial-to-mesenchymal transition in human aortic endothelial cells
por: Tang, Rining, et al.
Publicado: (2010) -
Interleukin-1β mediates high glucose induced phenotypic transition in human aortic endothelial cells
por: Zhu, Dong-Dong, et al.
Publicado: (2016) -
Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β–dependent vascular endothelial cadherin tyrosine phosphorylation
por: Haidari, Mehran, et al.
Publicado: (2014) -
Disruption of endothelial adherens junctions by high glucose is mediated by protein kinase C-β–dependent vascular endothelial cadherin tyrosine phosphorylation
por: Haidari, Mehran, et al.
Publicado: (2014) -
Propofol protects against high glucose-induced endothelial adhesion molecules expression in human umbilical vein endothelial cells
por: Zhu, Minmin, et al.
Publicado: (2013)