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Deep brain stimulation for refractory epilepsy

Deep brain stimulation (DBS) is a method of treatment utilized to control medically refractory epilepsy (RE). Patients with medically refractory epilepsy who do not achieve satisfactory control of seizures with pharmacological treatment or surgical resection of the epileptic focus and those who do n...

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Detalles Bibliográficos
Autores principales: Tykocki, Tomasz, Mandat, Tomasz, Kornakiewicz, Anna, Koziara, Henryk, Nauman, Paweł
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Termedia Publishing House 2012
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3506228/
https://www.ncbi.nlm.nih.gov/pubmed/23185188
http://dx.doi.org/10.5114/aoms.2012.31135
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author Tykocki, Tomasz
Mandat, Tomasz
Kornakiewicz, Anna
Koziara, Henryk
Nauman, Paweł
author_facet Tykocki, Tomasz
Mandat, Tomasz
Kornakiewicz, Anna
Koziara, Henryk
Nauman, Paweł
author_sort Tykocki, Tomasz
collection PubMed
description Deep brain stimulation (DBS) is a method of treatment utilized to control medically refractory epilepsy (RE). Patients with medically refractory epilepsy who do not achieve satisfactory control of seizures with pharmacological treatment or surgical resection of the epileptic focus and those who do not qualify for surgery could benefit from DBS. The most frequently used stereotactic targets for DBS are the anterior thalamic nucleus, subthalamic nucleus, central-medial thalamic nucleus, hippocampus, amygdala and cerebellum. The DBS is believed to be an effective method of treatment for various types of epilepsy among adults and adolescents. Side effects may be associated with implantation of electrodes and with the stimulation itself. An increasing number of publications and growing interest in DBS application for RE may result in standardization of the qualification and treatment protocol for RE with DBS.
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spelling pubmed-35062282012-11-26 Deep brain stimulation for refractory epilepsy Tykocki, Tomasz Mandat, Tomasz Kornakiewicz, Anna Koziara, Henryk Nauman, Paweł Arch Med Sci State of the Art Paper Deep brain stimulation (DBS) is a method of treatment utilized to control medically refractory epilepsy (RE). Patients with medically refractory epilepsy who do not achieve satisfactory control of seizures with pharmacological treatment or surgical resection of the epileptic focus and those who do not qualify for surgery could benefit from DBS. The most frequently used stereotactic targets for DBS are the anterior thalamic nucleus, subthalamic nucleus, central-medial thalamic nucleus, hippocampus, amygdala and cerebellum. The DBS is believed to be an effective method of treatment for various types of epilepsy among adults and adolescents. Side effects may be associated with implantation of electrodes and with the stimulation itself. An increasing number of publications and growing interest in DBS application for RE may result in standardization of the qualification and treatment protocol for RE with DBS. Termedia Publishing House 2012-10-08 2012-11-09 /pmc/articles/PMC3506228/ /pubmed/23185188 http://dx.doi.org/10.5114/aoms.2012.31135 Text en Copyright © 2012 Termedia & Banach http://creativecommons.org/licenses/by-nc-nd/3.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution-Noncommercial 3.0 Unported License, permitting all non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle State of the Art Paper
Tykocki, Tomasz
Mandat, Tomasz
Kornakiewicz, Anna
Koziara, Henryk
Nauman, Paweł
Deep brain stimulation for refractory epilepsy
title Deep brain stimulation for refractory epilepsy
title_full Deep brain stimulation for refractory epilepsy
title_fullStr Deep brain stimulation for refractory epilepsy
title_full_unstemmed Deep brain stimulation for refractory epilepsy
title_short Deep brain stimulation for refractory epilepsy
title_sort deep brain stimulation for refractory epilepsy
topic State of the Art Paper
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3506228/
https://www.ncbi.nlm.nih.gov/pubmed/23185188
http://dx.doi.org/10.5114/aoms.2012.31135
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