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Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome

A 32-year-old man with blurred vision in the right eye and headache presented with anterior uveitis, an intraocular pressure (IOP) of 60 mmHg, an open angle, no visual field defects, and normal optic nerve. He had a history of five previous similar attacks. In each of the previous instances, his ant...

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Detalles Bibliográficos
Autores principales: Kim, Tae-Hyup, Kim, Jung Lim, Kee, Changwon
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Korean Ophthalmological Society 2012
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3506825/
https://www.ncbi.nlm.nih.gov/pubmed/23204806
http://dx.doi.org/10.3341/kjo.2012.26.6.473
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author Kim, Tae-Hyup
Kim, Jung Lim
Kee, Changwon
author_facet Kim, Tae-Hyup
Kim, Jung Lim
Kee, Changwon
author_sort Kim, Tae-Hyup
collection PubMed
description A 32-year-old man with blurred vision in the right eye and headache presented with anterior uveitis, an intraocular pressure (IOP) of 60 mmHg, an open angle, no visual field defects, and normal optic nerve. He had a history of five previous similar attacks. In each of the previous instances, his anterior uveitis and high IOP were controlled with antiglaucoma medications and topical steroids. However, at the fifth attack, his optic disc was pale and a superior paracentral visual field defect was shown. Brain magnetic resonance image studies were normal. This case represents that a recurrent Posner-Schlossman syndrome (PSS)-induced optic disc atrophy likely due to ocular ischemia caused by a recurrent, high IOP. Although PSS is a self-limiting syndrome, we should manage high IOP and prevent ischemia of the optic nerve head by treating with ocular antihypertensive medications.
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spelling pubmed-35068252012-12-01 Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome Kim, Tae-Hyup Kim, Jung Lim Kee, Changwon Korean J Ophthalmol Case Report A 32-year-old man with blurred vision in the right eye and headache presented with anterior uveitis, an intraocular pressure (IOP) of 60 mmHg, an open angle, no visual field defects, and normal optic nerve. He had a history of five previous similar attacks. In each of the previous instances, his anterior uveitis and high IOP were controlled with antiglaucoma medications and topical steroids. However, at the fifth attack, his optic disc was pale and a superior paracentral visual field defect was shown. Brain magnetic resonance image studies were normal. This case represents that a recurrent Posner-Schlossman syndrome (PSS)-induced optic disc atrophy likely due to ocular ischemia caused by a recurrent, high IOP. Although PSS is a self-limiting syndrome, we should manage high IOP and prevent ischemia of the optic nerve head by treating with ocular antihypertensive medications. The Korean Ophthalmological Society 2012-12 2012-11-12 /pmc/articles/PMC3506825/ /pubmed/23204806 http://dx.doi.org/10.3341/kjo.2012.26.6.473 Text en © 2012 The Korean Ophthalmological Society http://creativecommons.org/licenses/by-nc/3.0/ This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/3.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Kim, Tae-Hyup
Kim, Jung Lim
Kee, Changwon
Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title_full Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title_fullStr Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title_full_unstemmed Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title_short Optic Disc Atrophy in Patient with Posner-Schlossman Syndrome
title_sort optic disc atrophy in patient with posner-schlossman syndrome
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3506825/
https://www.ncbi.nlm.nih.gov/pubmed/23204806
http://dx.doi.org/10.3341/kjo.2012.26.6.473
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