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Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients

We demonstrated that visfatin expressed in monocytes and neutrophils and increased their reactivity in male acute ST-segment elevation myocardial infarction patients. Furthermore, visfatin was strongly appeared in lipid rich coronary rupture plaques and macrophages. These results suggest another exp...

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Autores principales: Chiu, Cheng-An, Yu, Teng-Hung, Hung, Wei-Chin, Lu, Li-Fen, Chung, Fu-Mei, Tsai, I-Ting, Yang, Chih-Ying, Hsu, Chia-Chang, Lu, Yung-Chuan, Houng, Jer-Yiing, Lee, Yau-Jiunn, Wang, Chao-Ping
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2012
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3530866/
https://www.ncbi.nlm.nih.gov/pubmed/23304061
http://dx.doi.org/10.1155/2012/469852
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author Chiu, Cheng-An
Yu, Teng-Hung
Hung, Wei-Chin
Lu, Li-Fen
Chung, Fu-Mei
Tsai, I-Ting
Yang, Chih-Ying
Hsu, Chia-Chang
Lu, Yung-Chuan
Houng, Jer-Yiing
Lee, Yau-Jiunn
Wang, Chao-Ping
author_facet Chiu, Cheng-An
Yu, Teng-Hung
Hung, Wei-Chin
Lu, Li-Fen
Chung, Fu-Mei
Tsai, I-Ting
Yang, Chih-Ying
Hsu, Chia-Chang
Lu, Yung-Chuan
Houng, Jer-Yiing
Lee, Yau-Jiunn
Wang, Chao-Ping
author_sort Chiu, Cheng-An
collection PubMed
description We demonstrated that visfatin expressed in monocytes and neutrophils and increased their reactivity in male acute ST-segment elevation myocardial infarction patients. Furthermore, visfatin was strongly appeared in lipid rich coronary rupture plaques and macrophages. These results suggest another explanation about leukocytes mediated visfatin that may play a pathogenesis role in coronary vulnerable plaques rupture.
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spelling pubmed-35308662013-01-09 Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients Chiu, Cheng-An Yu, Teng-Hung Hung, Wei-Chin Lu, Li-Fen Chung, Fu-Mei Tsai, I-Ting Yang, Chih-Ying Hsu, Chia-Chang Lu, Yung-Chuan Houng, Jer-Yiing Lee, Yau-Jiunn Wang, Chao-Ping Mediators Inflamm Clinical Study We demonstrated that visfatin expressed in monocytes and neutrophils and increased their reactivity in male acute ST-segment elevation myocardial infarction patients. Furthermore, visfatin was strongly appeared in lipid rich coronary rupture plaques and macrophages. These results suggest another explanation about leukocytes mediated visfatin that may play a pathogenesis role in coronary vulnerable plaques rupture. Hindawi Publishing Corporation 2012 2012-12-13 /pmc/articles/PMC3530866/ /pubmed/23304061 http://dx.doi.org/10.1155/2012/469852 Text en Copyright © 2012 Cheng-An Chiu et al. https://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Clinical Study
Chiu, Cheng-An
Yu, Teng-Hung
Hung, Wei-Chin
Lu, Li-Fen
Chung, Fu-Mei
Tsai, I-Ting
Yang, Chih-Ying
Hsu, Chia-Chang
Lu, Yung-Chuan
Houng, Jer-Yiing
Lee, Yau-Jiunn
Wang, Chao-Ping
Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title_full Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title_fullStr Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title_full_unstemmed Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title_short Increased Expression of Visfatin in Monocytes and Macrophages in Male Acute Myocardial Infarction Patients
title_sort increased expression of visfatin in monocytes and macrophages in male acute myocardial infarction patients
topic Clinical Study
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3530866/
https://www.ncbi.nlm.nih.gov/pubmed/23304061
http://dx.doi.org/10.1155/2012/469852
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