Transformation of membranous into anti-GBM nephritis

Membranous nephropathy is a common glomerular disease. We report a 50-year-old man with a history of membranous nephropathy in remission, who presented with acute kidney injury, proteinuria, hematuria, and hypertension. He also had a high anti-glomerular basement membrane (anti-GBM) antibody titer a...

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Autores principales: Imtiaz, S., Alswaida, A., Rehman, H., Faraz, N., Afshan, T., Alkafoury, H., Qayyum, S., AL Khoiter, M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2012
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3544060/
https://www.ncbi.nlm.nih.gov/pubmed/23326049
http://dx.doi.org/10.4103/0971-4065.103924
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author Imtiaz, S.
Alswaida, A.
Rehman, H.
Faraz, N.
Afshan, T.
Alkafoury, H.
Qayyum, S.
AL Khoiter, M.
author_facet Imtiaz, S.
Alswaida, A.
Rehman, H.
Faraz, N.
Afshan, T.
Alkafoury, H.
Qayyum, S.
AL Khoiter, M.
author_sort Imtiaz, S.
collection PubMed
description Membranous nephropathy is a common glomerular disease. We report a 50-year-old man with a history of membranous nephropathy in remission, who presented with acute kidney injury, proteinuria, hematuria, and hypertension. He also had a high anti-glomerular basement membrane (anti-GBM) antibody titer and crescent transformation of primary pathology. The kidney functions deteriorated rapidly despite aggressive therapy with cyclophosphamide, methylprednisolone, and plasmapheresis.
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spelling pubmed-35440602013-01-16 Transformation of membranous into anti-GBM nephritis Imtiaz, S. Alswaida, A. Rehman, H. Faraz, N. Afshan, T. Alkafoury, H. Qayyum, S. AL Khoiter, M. Indian J Nephrol Case Report Membranous nephropathy is a common glomerular disease. We report a 50-year-old man with a history of membranous nephropathy in remission, who presented with acute kidney injury, proteinuria, hematuria, and hypertension. He also had a high anti-glomerular basement membrane (anti-GBM) antibody titer and crescent transformation of primary pathology. The kidney functions deteriorated rapidly despite aggressive therapy with cyclophosphamide, methylprednisolone, and plasmapheresis. Medknow Publications & Media Pvt Ltd 2012 /pmc/articles/PMC3544060/ /pubmed/23326049 http://dx.doi.org/10.4103/0971-4065.103924 Text en Copyright: © Indian Journal of Nephrology http://creativecommons.org/licenses/by-nc-sa/3.0 This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Imtiaz, S.
Alswaida, A.
Rehman, H.
Faraz, N.
Afshan, T.
Alkafoury, H.
Qayyum, S.
AL Khoiter, M.
Transformation of membranous into anti-GBM nephritis
title Transformation of membranous into anti-GBM nephritis
title_full Transformation of membranous into anti-GBM nephritis
title_fullStr Transformation of membranous into anti-GBM nephritis
title_full_unstemmed Transformation of membranous into anti-GBM nephritis
title_short Transformation of membranous into anti-GBM nephritis
title_sort transformation of membranous into anti-gbm nephritis
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3544060/
https://www.ncbi.nlm.nih.gov/pubmed/23326049
http://dx.doi.org/10.4103/0971-4065.103924
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