The myeloid response to pancreatic carcinogenesis is regulated by the receptor for advanced glycation end-products

We identified a critical role for receptor for advanced glycation end products (RAGE) in the intratumoral accumulation of myeloid-derived suppressor cells (MDSCs) during pancreatic carcinogenesis. The absence of RAGE markedly delayed neoplasia and limited MDSC accumulation in mice expressing an onco...

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Detalles Bibliográficos
Autores principales: Vernon, Philip J., Zeh III, Herbert J., Lotze, Michael T.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Landes Bioscience 2013
Materias:
Author's View
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3667906/
https://www.ncbi.nlm.nih.gov/pubmed/23762800
http://dx.doi.org/10.4161/onci.24184

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3667906/
https://www.ncbi.nlm.nih.gov/pubmed/23762800
http://dx.doi.org/10.4161/onci.24184

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