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Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely

Detalles Bibliográficos
Autor principal: Robinson, Richard
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Public Library of Science 2013
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3672213/
https://www.ncbi.nlm.nih.gov/pubmed/23750117
http://dx.doi.org/10.1371/journal.pbio.1001575
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spelling pubmed-36722132013-06-07 Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely Robinson, Richard PLoS Biol Synopsis Public Library of Science 2013-06-04 /pmc/articles/PMC3672213/ /pubmed/23750117 http://dx.doi.org/10.1371/journal.pbio.1001575 Text en © 2013 Richard Robinson http://creativecommons.org/licenses/by/4.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited.
spellingShingle Synopsis
Robinson, Richard
Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title_full Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title_fullStr Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title_full_unstemmed Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title_short Promote One, Inhibit the Other: A Single Pathway Controls Axon and Dendrite Growth, Oppositely
title_sort promote one, inhibit the other: a single pathway controls axon and dendrite growth, oppositely
topic Synopsis
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3672213/
https://www.ncbi.nlm.nih.gov/pubmed/23750117
http://dx.doi.org/10.1371/journal.pbio.1001575
work_keys_str_mv AT robinsonrichard promoteoneinhibittheotherasinglepathwaycontrolsaxonanddendritegrowthoppositely