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Mutations in Tyr(808) reveal a potential auto-inhibitory mechanism of guanylate cyclase-B regulation
In this study, Tyr(808) in GC-B (guanylate cyclase-B), a receptor of the CNP (C-type natriuretic peptide), has been shown to be a critical regulator of GC-B activity. In searching for phosphorylation sites that could account for suppression of GC-B activity by S1P (sphingosine-1-phosphate), mutation...
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
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Portland Press Ltd.
2013
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3673034/ https://www.ncbi.nlm.nih.gov/pubmed/23586811 http://dx.doi.org/10.1042/BSR20130025 |