Cargando…
Cytokine Receptor-Like Factor 1 (CRLF1) Protects against 6-Hydroxydopamine Toxicity Independent of the gp130/JAK Signaling Pathway
Oxidative stress is an important cause of cellular toxicity in the central nervous system and contributes to the pathology associated with neurodegenerative disorders including Parkinson’s disease. As such, elucidation of cellular mechanisms that enhance neuronal resistance to oxidative stress may p...
Autores principales: | Looyenga, Brendan D., Resau, James, MacKeigan, Jeffrey P. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3688593/ https://www.ncbi.nlm.nih.gov/pubmed/23818941 http://dx.doi.org/10.1371/journal.pone.0066548 |
Ejemplares similares
-
Correction: Cytokine Receptor-Like Factor 1 (CRLF1) Protects against 6-Hydroxydopamine Toxicity Independent of the gp130/JAK Signaling Pathway
por: Looyenga, Brendan D., et al.
Publicado: (2014) -
STAT3 Is Activated by JAK2 Independent of Key Oncogenic Driver Mutations in Non-Small Cell Lung Carcinoma
por: Looyenga, Brendan D., et al.
Publicado: (2012) -
The Role of gp130 Cytokines in Tuberculosis
por: Ritter, Kristina, et al.
Publicado: (2020) -
GP130 Cytokines in Breast Cancer and Bone
por: Omokehinde, Tolu, et al.
Publicado: (2020) -
Narrative-based computational modelling of the Gp130/JAK/STAT signalling pathway
por: Guerriero, Maria Luisa, et al.
Publicado: (2009)