Cargando…
Interactions between adenosine, angiotensin II and nitric oxide on the afferent arteriole influence sensitivity of the tubuloglomerular feedback
Adenosine, via activation of A(1) receptors on the afferent arteriole (AA), mediates the tubuloglomerular feedback (TGF) mechanism. Angiotensin II and nitric oxide (NO) can modulate the sensitivity of the TGF mechanism. However, the interaction among these substances in regulating the TGF resetting...
Autores principales: | Persson, A. E. G., Lai, En Yin, Gao, Xiang, Carlström, Mattias, Patzak, Andreas |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3714451/ https://www.ncbi.nlm.nih.gov/pubmed/23882224 http://dx.doi.org/10.3389/fphys.2013.00187 |
Ejemplares similares
-
Deficiency of Nox2 prevents angiotensin II-induced inward remodeling in cerebral arterioles
por: Chan, Siu-Lung, et al.
Publicado: (2013) -
Reduced tubuloglomerular feedback activity and absence of its synchronization in a connexin40 knockout rat
por: More, Heather L., et al.
Publicado: (2023) -
Lack of Connexins 40 and 45 Reduces Local and Conducted Vasoconstrictor Responses in the Murine Afferent Arterioles
por: Møller, Sophie, et al.
Publicado: (2020) -
Fetal tubuloglomerular feedback in an ovine model of mild maternal renal disease
por: Turner, Anita J, et al.
Publicado: (2015) -
Highly sensitive myosin phosphorylation analysis in the renal afferent
arteriole
por: Takeya, Kosuke
Publicado: (2016)