Expression of the NF-κB inhibitor ABIN-3 in response to TNF and toll-like receptor 4 stimulation is itself regulated by NF-κB

Although the nuclear factor-κB (NF-κB)-dependent gene expression is critical to the induction of an efficient immune response to infection or tissue injury, excessive or prolonged NF-κB signalling can contribute to the development of several inflammatory diseases. Therefore, the NF-κB signal transdu...

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Detalles Bibliográficos
Autores principales: Verstrepen, Lynn, Adib-Conquy, Minou, Kreike, Marja, Carpentier, Isabelle, Adrie, Christophe, Cavaillon, Jean-Marc, Beyaert, Rudi
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Blackwell Publishing Ltd 2008
Materias:
In Focus
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3823492/
https://www.ncbi.nlm.nih.gov/pubmed/18081698
http://dx.doi.org/10.1111/j.1582-4934.2007.00187.x

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