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Acute Inhibition of Excessive Mitochondrial Fission After Myocardial Infarction Prevents Long‐term Cardiac Dysfunction
BACKGROUND: Ischemia and reperfusion (IR) injury remains a major cause of morbidity and mortality and multiple molecular and cellular pathways have been implicated in this injury. We determined whether acute inhibition of excessive mitochondrial fission at the onset of reperfusion improves mitochond...
Autores principales: | Disatnik, Marie‐Hélène, Ferreira, Julio C.B., Campos, Juliane Cruz, Gomes, Kátia Sampaio, Dourado, Paulo M.M., Qi, Xin, Mochly‐Rosen, Daria |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Blackwell Publishing Ltd
2013
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3835263/ https://www.ncbi.nlm.nih.gov/pubmed/24103571 http://dx.doi.org/10.1161/JAHA.113.000461 |
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