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Hypokalemia-Induced Abnormal Movements: Case Report
INTRODUCTION: A sudden loss of consciousness followed by abnormal movements can be ictal or syncopal in origin. Transient response by the brain to sudden decrease of blood flow may cause sudden loss of consciousness followed by abnormal movements that mimic seizure. Dysrhythmia is one of the importa...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Kowsar
2013
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3864401/ https://www.ncbi.nlm.nih.gov/pubmed/24350174 http://dx.doi.org/10.5812/traumamon.12016 |
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author | Baratloo, Alireza Rouhipour, Alaleh Forouzanfar, Mohammadmahdi Rahmati, Farhad Hashemi, Behrooz |
author_facet | Baratloo, Alireza Rouhipour, Alaleh Forouzanfar, Mohammadmahdi Rahmati, Farhad Hashemi, Behrooz |
author_sort | Baratloo, Alireza |
collection | PubMed |
description | INTRODUCTION: A sudden loss of consciousness followed by abnormal movements can be ictal or syncopal in origin. Transient response by the brain to sudden decrease of blood flow may cause sudden loss of consciousness followed by abnormal movements that mimic seizure. Dysrhythmia is one of the important and critical reasons of such events that should be differentiated from seizure. CASE PRESENTATION: In this case report we describe a 55 year-old woman admitted to our emergency department first with the impression of seizure. Eventually, it was realized that she had suffered from brain hypo-perfusion secondary to hypokalemia induced arrhythmia. Her arrhythmia was managed by unsynchronized biphasic shock in acute phase and also potassium replacement. She was then admitted to the CCU (Coronary Care Unit) where she received further care for medical management and drug dose adjustment and was discharged 4 days later. CONCLUSIONS: Syncope from arrhythmia most commonly results from ventricular tachycardia, which accounts for 11% of all cases of syncope. Torsades de point is a unique type of ventricular tachycardia, characterized by QRS complexes of changing amplitude proceeded by prolonged QT intervals and almost often followed by loss of consciousness and also seizure like movements. Prolonged QT interval which is an important provocative factor for torsades de point commonly results from interactions between drug therapy, myocardial ischemia, and electrolyte disturbances such as hypokalemia or hypomagnesaemia. Changes in the extracellular potassium level have predominant and profound influences on the function of the cardiovascular system that may provoke fatal demonstrations such as QT prolongation, ventricular arrhythmia and even cardiac arrest. Electrolyte assessment is particularly important in certain patient populations, such as the elderly in whom a variety of pathological states or conditions like dehydration or renal failure are more common. Early identification and correction of these disturbances are necessary to control either seizures or seizure-like movements and prevent permanent brain damage, as anticonvulsants alone are generally ineffective. |
format | Online Article Text |
id | pubmed-3864401 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2013 |
publisher | Kowsar |
record_format | MEDLINE/PubMed |
spelling | pubmed-38644012013-12-17 Hypokalemia-Induced Abnormal Movements: Case Report Baratloo, Alireza Rouhipour, Alaleh Forouzanfar, Mohammadmahdi Rahmati, Farhad Hashemi, Behrooz Trauma Mon Case Report INTRODUCTION: A sudden loss of consciousness followed by abnormal movements can be ictal or syncopal in origin. Transient response by the brain to sudden decrease of blood flow may cause sudden loss of consciousness followed by abnormal movements that mimic seizure. Dysrhythmia is one of the important and critical reasons of such events that should be differentiated from seizure. CASE PRESENTATION: In this case report we describe a 55 year-old woman admitted to our emergency department first with the impression of seizure. Eventually, it was realized that she had suffered from brain hypo-perfusion secondary to hypokalemia induced arrhythmia. Her arrhythmia was managed by unsynchronized biphasic shock in acute phase and also potassium replacement. She was then admitted to the CCU (Coronary Care Unit) where she received further care for medical management and drug dose adjustment and was discharged 4 days later. CONCLUSIONS: Syncope from arrhythmia most commonly results from ventricular tachycardia, which accounts for 11% of all cases of syncope. Torsades de point is a unique type of ventricular tachycardia, characterized by QRS complexes of changing amplitude proceeded by prolonged QT intervals and almost often followed by loss of consciousness and also seizure like movements. Prolonged QT interval which is an important provocative factor for torsades de point commonly results from interactions between drug therapy, myocardial ischemia, and electrolyte disturbances such as hypokalemia or hypomagnesaemia. Changes in the extracellular potassium level have predominant and profound influences on the function of the cardiovascular system that may provoke fatal demonstrations such as QT prolongation, ventricular arrhythmia and even cardiac arrest. Electrolyte assessment is particularly important in certain patient populations, such as the elderly in whom a variety of pathological states or conditions like dehydration or renal failure are more common. Early identification and correction of these disturbances are necessary to control either seizures or seizure-like movements and prevent permanent brain damage, as anticonvulsants alone are generally ineffective. Kowsar 2013-10-13 2013-12 /pmc/articles/PMC3864401/ /pubmed/24350174 http://dx.doi.org/10.5812/traumamon.12016 Text en Copyright © 2013, Trauma Research Center http://creativecommons.org/licenses/by/3.0 This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Case Report Baratloo, Alireza Rouhipour, Alaleh Forouzanfar, Mohammadmahdi Rahmati, Farhad Hashemi, Behrooz Hypokalemia-Induced Abnormal Movements: Case Report |
title | Hypokalemia-Induced Abnormal Movements: Case Report |
title_full | Hypokalemia-Induced Abnormal Movements: Case Report |
title_fullStr | Hypokalemia-Induced Abnormal Movements: Case Report |
title_full_unstemmed | Hypokalemia-Induced Abnormal Movements: Case Report |
title_short | Hypokalemia-Induced Abnormal Movements: Case Report |
title_sort | hypokalemia-induced abnormal movements: case report |
topic | Case Report |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3864401/ https://www.ncbi.nlm.nih.gov/pubmed/24350174 http://dx.doi.org/10.5812/traumamon.12016 |
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