Cargando…
TRAF1 is a critical regulator of cerebral ischaemia–reperfusion injury and neuronal death
Stroke is a leading global cause of mortality and disability. Less than 5% of patients are able to receive tissue plasminogen activator thrombolysis within the necessary timeframe. Focusing on the process of neuronal apoptosis in the penumbra, which lasts from hours to days after ischaemia, appears...
Autores principales: | Lu, Yan-Yun, Li, Zuo-Zhi, Jiang, Ding-Sheng, Wang, Lang, Zhang, Yan, Chen, Ke, Zhang, Xiao-Fei, Liu, Yi, Fan, Guo-Chang, Chen, Yingjie, Yang, Qinglin, Zhou, Yan, Zhang, Xiao-Dong, Liu, De-Pei, Li, Hongliang |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Pub. Group
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3868160/ https://www.ncbi.nlm.nih.gov/pubmed/24284943 http://dx.doi.org/10.1038/ncomms3852 |
Ejemplares similares
-
β‐arrestin 2 negatively regulates NOD2 signalling pathway through association with TRAF6 in microglia after cerebral ischaemia/reperfusion injury
por: Chen, Lin, et al.
Publicado: (2019) -
HDAC9 exacerbates endothelial injury in cerebral ischaemia/reperfusion injury
por: Shi, Weichen, et al.
Publicado: (2016) -
Mitochondrial ferritin attenuates cerebral ischaemia/reperfusion injury by inhibiting ferroptosis
por: Wang, Peina, et al.
Publicado: (2021) -
Inhibition of autophagy‐dependent pyroptosis attenuates cerebral ischaemia/reperfusion injury
por: Liu, Hui, et al.
Publicado: (2021) -
XingNaoJing injection ameliorates cerebral ischaemia/reperfusion injury via SIRT1-mediated inflammatory response inhibition
por: Zhang, Yue-Ming, et al.
Publicado: (2019)