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p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)

Developmental endothelial locus-1 (Del-1) is an endothelium-derived anti-inflammatory molecule that is downregulated by inflammatory stimuli. Little is known about the molecular mechanisms by which Del-1 transcription is regulated. In the present study, a DNA sequence upstream of the Del-1 gene was...

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Detalles Bibliográficos
Autores principales: Kim, Hyesoon, Lee, Seung-Hwan, Lee, Mi-Ni, Oh, Goo Taeg, Choi, Kyung-Chul, Choi, Eun Young
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Impact Journals LLC 2013
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3875763/
https://www.ncbi.nlm.nih.gov/pubmed/24192518
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author Kim, Hyesoon
Lee, Seung-Hwan
Lee, Mi-Ni
Oh, Goo Taeg
Choi, Kyung-Chul
Choi, Eun Young
author_facet Kim, Hyesoon
Lee, Seung-Hwan
Lee, Mi-Ni
Oh, Goo Taeg
Choi, Kyung-Chul
Choi, Eun Young
author_sort Kim, Hyesoon
collection PubMed
description Developmental endothelial locus-1 (Del-1) is an endothelium-derived anti-inflammatory molecule that is downregulated by inflammatory stimuli. Little is known about the molecular mechanisms by which Del-1 transcription is regulated. In the present study, a DNA sequence upstream of the Del-1 gene was analyzed and putative p53 response elements (p53REs) were identified. An approximately 2 kb fragment upstream of the translation start site displayed the highest Del-1 transcriptional activity, and the transcriptional activity of this fragment was enhanced by overexpression of p53. Chemical activation of endogenous p53 elevated the levels of Del-1 mRNA. Site-directed mutagenesis of CATG in the consensus sequences of the 2 kb fragment to TATA significantly reduced the transcription of Del-1. Chromatin immunoprecipitation revealed recruitment of p53 to the p53REs of the Del-1 promoter, resulting in increased Del-1 transcription. Finally, primary endothelial cells isolated from mice with reduced levels of p53 showed a decrease in Del-1 mRNA compared to wild-type endothelial cells. Moreover, Del-1 reciprocally enhanced p53 expression in primary endothelial cells. Thus, these findings suggest that Del-1 is a novel transcriptional target gene of p53.
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spelling pubmed-38757632014-01-07 p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1) Kim, Hyesoon Lee, Seung-Hwan Lee, Mi-Ni Oh, Goo Taeg Choi, Kyung-Chul Choi, Eun Young Oncotarget Research Paper Developmental endothelial locus-1 (Del-1) is an endothelium-derived anti-inflammatory molecule that is downregulated by inflammatory stimuli. Little is known about the molecular mechanisms by which Del-1 transcription is regulated. In the present study, a DNA sequence upstream of the Del-1 gene was analyzed and putative p53 response elements (p53REs) were identified. An approximately 2 kb fragment upstream of the translation start site displayed the highest Del-1 transcriptional activity, and the transcriptional activity of this fragment was enhanced by overexpression of p53. Chemical activation of endogenous p53 elevated the levels of Del-1 mRNA. Site-directed mutagenesis of CATG in the consensus sequences of the 2 kb fragment to TATA significantly reduced the transcription of Del-1. Chromatin immunoprecipitation revealed recruitment of p53 to the p53REs of the Del-1 promoter, resulting in increased Del-1 transcription. Finally, primary endothelial cells isolated from mice with reduced levels of p53 showed a decrease in Del-1 mRNA compared to wild-type endothelial cells. Moreover, Del-1 reciprocally enhanced p53 expression in primary endothelial cells. Thus, these findings suggest that Del-1 is a novel transcriptional target gene of p53. Impact Journals LLC 2013-10-03 /pmc/articles/PMC3875763/ /pubmed/24192518 Text en Copyright: © 2013 Kim et al. http://creativecommons.org/licenses/by/2.5/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited
spellingShingle Research Paper
Kim, Hyesoon
Lee, Seung-Hwan
Lee, Mi-Ni
Oh, Goo Taeg
Choi, Kyung-Chul
Choi, Eun Young
p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title_full p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title_fullStr p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title_full_unstemmed p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title_short p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (Del-1)
title_sort p53 regulates the transcription of the anti-inflammatory molecule developmental endothelial locus-1 (del-1)
topic Research Paper
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3875763/
https://www.ncbi.nlm.nih.gov/pubmed/24192518
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