Cargando…
T-type Ca(2+) channels regulate the exit of cardiac myocytes from the cell cycle after birth
T-type Ca(2+) channels (TTCCs) are expressed in the fetal heart and then disappear from ventricular myocytes after birth. The hypothesis examined in this study was the α1G TTCCs' influence in myocyte maturation and their rapid withdrawal from the cell cycle after birth. METHODS: Cardiac myocyte...
Autores principales: | Wang, Fang, Gao, Hui, Kubo, Hajime, Fan, Xiaoxuan, Zhang, Hongyu, Berretta, Remus, Chen, Xiongwen, Sharp, Thomas, Starosta, Timothy, Makarewich, Catherine, Li, Ying, Molkentin, Jeffrey D., Houser, Steven R. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2013
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3888788/ https://www.ncbi.nlm.nih.gov/pubmed/23743021 http://dx.doi.org/10.1016/j.yjmcc.2013.05.016 |
Ejemplares similares
-
A low voltage activated Ca(2+) current found in a subset of human ventricular myocytes
por: Zhang, Xin, et al.
Publicado: (2020) -
Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection Fraction
por: Sharp, Thomas E., et al.
Publicado: (2017) -
Proarrhythmic Remodeling of Atrial Myocyte Ca(2+) Handling in Atrial Fibrillation
por: Houser, Steven R.
Publicado: (2023) -
Combining three independent pathological stressors induces a heart failure with preserved ejection fraction phenotype
por: Li, Yijia, et al.
Publicado: (2023) -
Ca(2+)-dependent modulation of voltage-gated myocyte sodium channels
por: Salvage, Samantha C., et al.
Publicado: (2021)