Cargando…
The anti-atherogenic cytokine interleukin-33 inhibits the expression of a disintegrin and metalloproteinase with thrombospondin motifs-1, -4 and -5 in human macrophages: Requirement of extracellular signal-regulated kinase, c-Jun N-terminal kinase and phosphoinositide 3-kinase signaling pathways()
Atherosclerosis is an inflammatory disorder of the vasculature regulated by cytokines. Amongst the cytokines, IL-33 attenuates the development of atherosclerosis in mouse model systems via several mechanisms, including inhibition of macrophage foam cell formation and promotion of a Th1 to Th2 shift....
Autores principales: | Ashlin, Tim G., Buckley, Melanie L., Salter, Rebecca C., Johnson, Jason L., Kwan, Alvin P.L., Ramji, Dipak P. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3928996/ https://www.ncbi.nlm.nih.gov/pubmed/24275094 http://dx.doi.org/10.1016/j.biocel.2013.11.008 |
Ejemplares similares
-
The expression of a disintegrin and metalloproteinase with thrombospondin motifs 4 in human macrophages is inhibited by the anti-atherogenic cytokine transforming growth factor-β and requires Smads, p38 mitogen-activated protein kinase and c-Jun
por: Salter, Rebecca C., et al.
Publicado: (2011) -
A novel role for c-Jun N-terminal kinase and phosphoinositide 3-kinase in the liver X receptor-mediated induction of macrophage gene expression
por: Huwait, Etimad A., et al.
Publicado: (2011) -
The interleukin-33-mediated inhibition of expression of two key genes implicated in atherosclerosis in human macrophages requires MAP kinase, phosphoinositide 3-kinase and nuclear factor-κB signaling pathways
por: Buckley, Melanie L., et al.
Publicado: (2019) -
The ADAMTS (A Disintegrin and Metalloproteinase with Thrombospondin motifs) family
por: Kelwick, Richard, et al.
Publicado: (2015) -
The Phosphoinositide 3-Kinase Signaling Pathway is Involved in the Control of Modified Low-Density Lipoprotein Uptake by Human Macrophages
por: Michael, Daryn R., et al.
Publicado: (2015)