Cargando…
Activation of peripheral KCNQ channels attenuates inflammatory pain
BACKGROUND: Refractory chronic pain dramatically reduces the quality of life of patients. Existing drugs cannot fully achieve effective chronic pain control because of their lower efficacy and/or accompanying side effects. Voltage-gated potassium channels (KCNQ) openers have demonstrated their analg...
Autores principales: | Hayashi, Hiroki, Iwata, Masashi, Tsuchimori, Noboru, Matsumoto, Tatsumi |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3936840/ https://www.ncbi.nlm.nih.gov/pubmed/24555569 http://dx.doi.org/10.1186/1744-8069-10-15 |
Ejemplares similares
-
Activation of peripheral KCNQ channels relieves gout pain
por: Zheng, Yueming, et al.
Publicado: (2015) -
Paclitaxel Inhibits KCNQ Channels in Primary Sensory Neurons to Initiate the Development of Painful Peripheral Neuropathy
por: Wu, Zizhen, et al.
Publicado: (2022) -
KCNQ2 and KCNQ5 form heteromeric channels independent of KCNQ3
por: Soh, Heun, et al.
Publicado: (2022) -
Activation of voltage-gated KCNQ/Kv7 channels by anticonvulsant retigabine attenuates mechanical allodynia of inflammatory temporomandibular joint in rats
por: Xu, Wen, et al.
Publicado: (2010) -
Drosophila KCNQ Channel Displays Evolutionarily Conserved Electrophysiology and Pharmacology with Mammalian KCNQ Channels
por: Cavaliere, Sonia, et al.
Publicado: (2011)