Wernicke's encephalopathy following hyperemesis gravidarum

Wernicke's encephalopathy (WE) is a potentially reversible yet serious neurological manifestation caused by vitamin B(1)(thiamine) deficiency. It is commonly associated with heavy alcohol consumption. Other clinical associations are with hyperemesis gravidarum (HG), starvation, and prolonged in...

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Autores principales: Kantor, Sandeep, Prakash, Sadanandan, Chandwani, Juhi, Gokhale, Antara, Sarma, Kalpana, Albahrani, Maher J.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2014
Materias:
Case Report
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3963199/
https://www.ncbi.nlm.nih.gov/pubmed/24701066
http://dx.doi.org/10.4103/0972-5229.128706
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author Kantor, Sandeep
Prakash, Sadanandan
Chandwani, Juhi
Gokhale, Antara
Sarma, Kalpana
Albahrani, Maher J.
author_facet Kantor, Sandeep
Prakash, Sadanandan
Chandwani, Juhi
Gokhale, Antara
Sarma, Kalpana
Albahrani, Maher J.
author_sort Kantor, Sandeep
collection PubMed
description Wernicke's encephalopathy (WE) is a potentially reversible yet serious neurological manifestation caused by vitamin B(1)(thiamine) deficiency. It is commonly associated with heavy alcohol consumption. Other clinical associations are with hyperemesis gravidarum (HG), starvation, and prolonged intravenous feeding. Most patients present with the triad of ocular signs, ataxia, and confusion. It can be associated with life-threatening complication like central pontine myelinolysis (CPM). We report two cases of WE following HG, with two different outcomes.
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spelling pubmed-39631992014-04-03 Wernicke's encephalopathy following hyperemesis gravidarum Kantor, Sandeep Prakash, Sadanandan Chandwani, Juhi Gokhale, Antara Sarma, Kalpana Albahrani, Maher J. Indian J Crit Care Med Case Report Wernicke's encephalopathy (WE) is a potentially reversible yet serious neurological manifestation caused by vitamin B(1)(thiamine) deficiency. It is commonly associated with heavy alcohol consumption. Other clinical associations are with hyperemesis gravidarum (HG), starvation, and prolonged intravenous feeding. Most patients present with the triad of ocular signs, ataxia, and confusion. It can be associated with life-threatening complication like central pontine myelinolysis (CPM). We report two cases of WE following HG, with two different outcomes. Medknow Publications & Media Pvt Ltd 2014-03 /pmc/articles/PMC3963199/ /pubmed/24701066 http://dx.doi.org/10.4103/0972-5229.128706 Text en Copyright: © Indian Journal of Critical Care Medicine http://creativecommons.org/licenses/by-nc-sa/3.0 This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Kantor, Sandeep
Prakash, Sadanandan
Chandwani, Juhi
Gokhale, Antara
Sarma, Kalpana
Albahrani, Maher J.
Wernicke's encephalopathy following hyperemesis gravidarum
title Wernicke's encephalopathy following hyperemesis gravidarum
title_full Wernicke's encephalopathy following hyperemesis gravidarum
title_fullStr Wernicke's encephalopathy following hyperemesis gravidarum
title_full_unstemmed Wernicke's encephalopathy following hyperemesis gravidarum
title_short Wernicke's encephalopathy following hyperemesis gravidarum
title_sort wernicke's encephalopathy following hyperemesis gravidarum
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3963199/
https://www.ncbi.nlm.nih.gov/pubmed/24701066
http://dx.doi.org/10.4103/0972-5229.128706
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