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Mitochondrial trafficking and anchoring in neurons: New insight and implications

Mitochondria are essential organelles for neuronal growth, survival, and function. Neurons use specialized mechanisms to drive mitochondria transport and to anchor them in axons and at synapses. Stationary mitochondria buffer intracellular Ca(2+) and serve as a local energy source by supplying ATP....

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Detalles Bibliográficos
Autor principal: Sheng, Zu-Hang
Formato: Online Artículo Texto
Lenguaje:English
Publicado: The Rockefeller University Press 2014
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3971748/
https://www.ncbi.nlm.nih.gov/pubmed/24687278
http://dx.doi.org/10.1083/jcb.201312123
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author Sheng, Zu-Hang
author_facet Sheng, Zu-Hang
author_sort Sheng, Zu-Hang
collection PubMed
description Mitochondria are essential organelles for neuronal growth, survival, and function. Neurons use specialized mechanisms to drive mitochondria transport and to anchor them in axons and at synapses. Stationary mitochondria buffer intracellular Ca(2+) and serve as a local energy source by supplying ATP. The balance between motile and stationary mitochondria responds quickly to changes in axonal and synaptic physiology. Defects in mitochondrial transport are implicated in the pathogenesis of several major neurological disorders. Recent work has provided new insight in the regulation of microtubule-based mitochondrial trafficking and anchoring, and on how mitochondrial motility influences neuron growth, synaptic function, and mitophagy.
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spelling pubmed-39717482014-10-01 Mitochondrial trafficking and anchoring in neurons: New insight and implications Sheng, Zu-Hang J Cell Biol Reviews Mitochondria are essential organelles for neuronal growth, survival, and function. Neurons use specialized mechanisms to drive mitochondria transport and to anchor them in axons and at synapses. Stationary mitochondria buffer intracellular Ca(2+) and serve as a local energy source by supplying ATP. The balance between motile and stationary mitochondria responds quickly to changes in axonal and synaptic physiology. Defects in mitochondrial transport are implicated in the pathogenesis of several major neurological disorders. Recent work has provided new insight in the regulation of microtubule-based mitochondrial trafficking and anchoring, and on how mitochondrial motility influences neuron growth, synaptic function, and mitophagy. The Rockefeller University Press 2014-03-31 /pmc/articles/PMC3971748/ /pubmed/24687278 http://dx.doi.org/10.1083/jcb.201312123 Text en This article is distributed under the terms of an Attribution–Noncommercial–Share Alike–No Mirror Sites license for the first six months after the publication date (see http://www.rupress.org/terms). After six months it is available under a Creative Commons License (Attribution–Noncommercial–Share Alike 3.0 Unported license, as described at http://creativecommons.org/licenses/by-nc-sa/3.0/).
spellingShingle Reviews
Sheng, Zu-Hang
Mitochondrial trafficking and anchoring in neurons: New insight and implications
title Mitochondrial trafficking and anchoring in neurons: New insight and implications
title_full Mitochondrial trafficking and anchoring in neurons: New insight and implications
title_fullStr Mitochondrial trafficking and anchoring in neurons: New insight and implications
title_full_unstemmed Mitochondrial trafficking and anchoring in neurons: New insight and implications
title_short Mitochondrial trafficking and anchoring in neurons: New insight and implications
title_sort mitochondrial trafficking and anchoring in neurons: new insight and implications
topic Reviews
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3971748/
https://www.ncbi.nlm.nih.gov/pubmed/24687278
http://dx.doi.org/10.1083/jcb.201312123
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