Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice

BACKGROUND: Traumatic brain injury (TBI) enhances pro-inflammatory responses, neuronal loss and long-term behavioral deficits. Caveolins (Cavs) are regulators of neuronal and glial survival signaling. Previously we showed that astrocyte and microglial activation is increased in Cav-1 knock-out (KO)...

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Detalles Bibliográficos
Autores principales: Niesman, Ingrid R, Schilling, Jan M, Shapiro, Lee A, Kellerhals, Sarah E, Bonds, Jacqueline A, Kleschevnikov, Alexander M, Cui, Weihua, Voong, April, Krajewski, Stan, Ali, Sameh S, Roth, David M, Patel, Hemal H, Patel, Piyush M, Head, Brian P
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2014
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3975903/
https://www.ncbi.nlm.nih.gov/pubmed/24593993
http://dx.doi.org/10.1186/1742-2094-11-39

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