Cargando…
Hypoperfusion, Mitochondria Failure, Oxidative Stress, and Alzheimer Disease
Autores principales: | Aliev, Gjumrakch, Obrenovich, Mark E., Smith, Mark A., Perry, George |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Hindawi Publishing Corporation
2003
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC400214/ https://www.ncbi.nlm.nih.gov/pubmed/12975531 http://dx.doi.org/10.1155/S1110724303305029 |
Ejemplares similares
-
Oxidative Stress Induced Mitochondrial Failure and Vascular Hypoperfusion as a Key Initiator for the Development of Alzheimer Disease
por: Aliev, Gjumrakch, et al.
Publicado: (2010) -
Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels: Implication in the pathogenesis of Alzheimer’s disease
por: Aliev, Gjumrakch, et al.
Publicado: (2008) -
Link between Cancer and Alzheimer Disease via Oxidative Stress Induced by Nitric Oxide-Dependent Mitochondrial DNA Overproliferation and Deletion
por: Aliev, Gjumrakch, et al.
Publicado: (2013) -
The GRK2 Overexpression Is a Primary Hallmark of Mitochondrial Lesions during Early Alzheimer Disease
por: Obrenovich, Mark E., et al.
Publicado: (2009) -
Insights into cerebrovascular complications and Alzheimer disease through the selective loss of GRK2 regulation
por: Obrenovich, Mark E, et al.
Publicado: (2009)