Cargando…
IL-17 induces production of IL-6 and IL-8 in rheumatoid arthritis synovial fibroblasts via NF-κB- and PI3-kinase/Akt-dependent pathways
Recent studies of the pathogenesis of rheumatoid arthritis (RA) have revealed that both synovial fibroblasts and T cells participate in the perpetuation of joint inflammation as dynamic partners in a mutual activation feedback, via secretion of cytokines and chemokines that stimulate each other. In...
Autores principales: | Hwang, Sue-Yun, Kim, Ju-Young, Kim, Kyoung-Woon, Park, Mi-Kyung, Moon, Youngmee, Kim, Wan-Uk, Kim, Ho-Youn |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2004
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC400429/ https://www.ncbi.nlm.nih.gov/pubmed/15059275 http://dx.doi.org/10.1186/ar1038 |
Ejemplares similares
-
IL-32 and IL-17 interact and have the potential to aggravate osteoclastogenesis in rheumatoid arthritis
por: Moon, Young-Mee, et al.
Publicado: (2012) -
Thrombospondin 2 Promotes IL-6 Production in Osteoarthritis Synovial Fibroblasts via the PI3K/AKT/NF-κB Pathway
por: Hou, Chun-Han, et al.
Publicado: (2021) -
IL-1 but not IL-18 induces osteoprotegerin and TRAIL in rheumatoid arthritis synovial fibroblasts
por: Morel, J, et al.
Publicado: (2003) -
IL-17-mediated mitochondrial dysfunction impairs apoptosis in rheumatoid arthritis synovial fibroblasts through activation of autophagy
por: Kim, Eun Kyung, et al.
Publicado: (2017) -
Expression levels of IL-15 and IL-17 in synovial fluid of rheumatoid arthritis animal model
por: Jin, Yi, et al.
Publicado: (2018)