Cargando…
Slc26a7 Chloride Channel Activity and Localization in Mouse Reissner’s Membrane Epithelium
Several members of the SLC26 gene family have highly-restricted expression patterns in the auditory and vestibular periphery and mutations in mice of at least two of these (SLC26A4 and SLC26A5) lead to deficits in hearing and/or balance. A previous report pointed to SLC26A7 as a candidate gene impor...
Autores principales: | Kim, Kyunghee X., Sanneman, Joel D., Kim, Hyoung-Mi, Harbidge, Donald G., Xu, Jie, Soleimani, Manoocher, Wangemann, Philine, Marcus, Daniel C. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4014619/ https://www.ncbi.nlm.nih.gov/pubmed/24810589 http://dx.doi.org/10.1371/journal.pone.0097191 |
Ejemplares similares
-
SLC26A4 Targeted to the Endolymphatic Sac Rescues Hearing and Balance in Slc26a4 Mutant Mice
por: Li, Xiangming, et al.
Publicado: (2013) -
Sodium selectivity of Reissner's membrane epithelial cells
por: Yamazaki, Muneharu, et al.
Publicado: (2011) -
Endolymphatic Na(+) and K(+) Concentrations during Cochlear Growth and Enlargement in Mice Lacking Slc26a4/pendrin
por: Li, Xiangming, et al.
Publicado: (2013) -
Deletion of Slc26a1 and Slc26a7 Delays Enamel Mineralization in Mice
por: Yin, Kaifeng, et al.
Publicado: (2017) -
Failure of Fluid Absorption in the Endolymphatic Sac Initiates Cochlear Enlargement that Leads to Deafness in Mice Lacking Pendrin Expression
por: Kim, Hyoung-Mi, et al.
Publicado: (2010)