A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact

The concept of the triad regulatory metasystem, which includes the neuroendocrine and immune regulation systems, is currently generally accepted. Changes occurring in each of the regulatory systems in response to the impact of technogenic chemical factors are also well known. This paper presents mat...

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Autores principales: Zaitseva, N. V., Kiryanov, D. A., Lanin, D. V., Chigvintsev, V. M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2014
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4020569/
https://www.ncbi.nlm.nih.gov/pubmed/24872840
http://dx.doi.org/10.1155/2014/492489
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author Zaitseva, N. V.
Kiryanov, D. A.
Lanin, D. V.
Chigvintsev, V. M.
author_facet Zaitseva, N. V.
Kiryanov, D. A.
Lanin, D. V.
Chigvintsev, V. M.
author_sort Zaitseva, N. V.
collection PubMed
description The concept of the triad regulatory metasystem, which includes the neuroendocrine and immune regulation systems, is currently generally accepted. Changes occurring in each of the regulatory systems in response to the impact of technogenic chemical factors are also well known. This paper presents mathematical models of the immune and neuroendocrine system functioning, using the interaction between these systems in response to bacterial invasion as an example, and changes in their performance under exposure to chemical factors, taking into account the stage of functional disorders in a producing organ, using the performance of the bone marrow as an example.
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spelling pubmed-40205692014-05-28 A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact Zaitseva, N. V. Kiryanov, D. A. Lanin, D. V. Chigvintsev, V. M. Comput Math Methods Med Research Article The concept of the triad regulatory metasystem, which includes the neuroendocrine and immune regulation systems, is currently generally accepted. Changes occurring in each of the regulatory systems in response to the impact of technogenic chemical factors are also well known. This paper presents mathematical models of the immune and neuroendocrine system functioning, using the interaction between these systems in response to bacterial invasion as an example, and changes in their performance under exposure to chemical factors, taking into account the stage of functional disorders in a producing organ, using the performance of the bone marrow as an example. Hindawi Publishing Corporation 2014 2014-04-27 /pmc/articles/PMC4020569/ /pubmed/24872840 http://dx.doi.org/10.1155/2014/492489 Text en Copyright © 2014 N. V. Zaitseva et al. https://creativecommons.org/licenses/by/3.0/ This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Research Article
Zaitseva, N. V.
Kiryanov, D. A.
Lanin, D. V.
Chigvintsev, V. M.
A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title_full A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title_fullStr A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title_full_unstemmed A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title_short A Mathematical Model of the Immune and Neuroendocrine Systems Mutual Regulation under the Technogenic Chemical Factors Impact
title_sort mathematical model of the immune and neuroendocrine systems mutual regulation under the technogenic chemical factors impact
topic Research Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4020569/
https://www.ncbi.nlm.nih.gov/pubmed/24872840
http://dx.doi.org/10.1155/2014/492489
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