Cargando…
Deficient repair response of IPF fibroblasts in a co-culture model of epithelial injury and repair
BACKGROUND: Idiopathic pulmonary fibrosis (IPF) is a progressive disorder marked by relentless fibrosis and damage of the lung architecture. A growing body of evidence now suggests that IPF progresses as a result of aberrant epithelial-fibroblast crosstalk. Injured epithelia are a major source of gr...
Autores principales: | Prasad, Sony, Hogaboam, Cory M, Jarai, Gabor |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4021590/ https://www.ncbi.nlm.nih.gov/pubmed/24834127 http://dx.doi.org/10.1186/1755-1536-7-7 |
Ejemplares similares
-
Senescent Cells in IPF: Locked in Repair?
por: Meiners, Silke, et al.
Publicado: (2020) -
Mitochondrial dysfunction contributes to the senescent phenotype of IPF lung fibroblasts
por: Schuliga, Michael, et al.
Publicado: (2018) -
Regeneration or Repair? The Role of Alveolar Epithelial Cells in the Pathogenesis of Idiopathic Pulmonary Fibrosis (IPF)
por: Confalonieri, Paola, et al.
Publicado: (2022) -
In vitro co-culture studies and the crucial role of fibroblast-immune cell crosstalk in IPF pathogenesis
por: Thiam, Fama, et al.
Publicado: (2023) -
Epithelial-fibroblast interactions in bleomycin-induced lung injury and repair.
por: Young, L, et al.
Publicado: (1993)