Cargando…
Pulmonary Hypertension in Wild Type Mice and Animals with Genetic Deficit in K(Ca)2.3 and K(Ca)3.1 Channels
OBJECTIVE: In vascular biology, endothelial K(Ca)2.3 and K(Ca)3.1 channels contribute to arterial blood pressure regulation by producing membrane hyperpolarization and smooth muscle relaxation. The role of K(Ca)2.3 and K(Ca)3.1 channels in the pulmonary circulation is not fully established. Using mi...
Autores principales: | Wandall-Frostholm, Christine, Skaarup, Lykke Moran, Sadda, Veeranjaneyulu, Nielsen, Gorm, Hedegaard, Elise Røge, Mogensen, Susie, Köhler, Ralf, Simonsen, Ulf |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4032241/ https://www.ncbi.nlm.nih.gov/pubmed/24858807 http://dx.doi.org/10.1371/journal.pone.0097687 |
Ejemplares similares
-
Down-regulation of K(Ca)2.3 channels causes erectile dysfunction in mice
por: Comerma-Steffensen, Simon, et al.
Publicado: (2017) -
Cancer-Associated Intermediate Conductance Ca(2+)-Activated K(+) Channel K(Ca)3.1
por: Mohr, Corinna J., et al.
Publicado: (2019) -
K(Ca)2 and K(Ca)3.1 Channels in the Airways: A New Therapeutic Target
por: Orfali, Razan, et al.
Publicado: (2023) -
Internalization and Transportation of Endothelial Cell Surface K(Ca)2.3 and K(Ca)3.1 in Normal Pregnancy and Preeclampsia
por: Choi, Shinkyu, et al.
Publicado: (2019) -
K(Ca)3.1 Channels and Glioblastoma: In Vitro Studies
por: Klumpp, Lukas, et al.
Publicado: (2018)