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Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis

Guillain-Barre syndrome (GBS) is a heterogenous group of peripheral-nerve disorders with similar clinical presentation characterized by acute, self-limited, progressive, bilateral and relatively symmetric ascending flaccid paralysis, which peaks in 2-4 weeks and then subsides. The usual complication...

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Detalles Bibliográficos
Autores principales: Saxena, Amrish, Singh, Vineeta, Verma, Nitin
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Medknow Publications & Media Pvt Ltd 2014
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4033859/
https://www.ncbi.nlm.nih.gov/pubmed/24872655
http://dx.doi.org/10.4103/0972-5229.130577
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author Saxena, Amrish
Singh, Vineeta
Verma, Nitin
author_facet Saxena, Amrish
Singh, Vineeta
Verma, Nitin
author_sort Saxena, Amrish
collection PubMed
description Guillain-Barre syndrome (GBS) is a heterogenous group of peripheral-nerve disorders with similar clinical presentation characterized by acute, self-limited, progressive, bilateral and relatively symmetric ascending flaccid paralysis, which peaks in 2-4 weeks and then subsides. The usual complications, which occur in a patient of GBS are pneumonia, sepsis, pulmonary embolism, respiratory insufficiency and cardiac arrest. The clinical course of GBS complicated by acute rhabdomyolysis is extremely rare. We present the case of GBS with marked elevation in serum creatine kinase, serum myoglobin levels and persistent hyperkalemia as a result of associated acute rhabdomyolysis.
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spelling pubmed-40338592014-05-28 Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis Saxena, Amrish Singh, Vineeta Verma, Nitin Indian J Crit Care Med Case Report Guillain-Barre syndrome (GBS) is a heterogenous group of peripheral-nerve disorders with similar clinical presentation characterized by acute, self-limited, progressive, bilateral and relatively symmetric ascending flaccid paralysis, which peaks in 2-4 weeks and then subsides. The usual complications, which occur in a patient of GBS are pneumonia, sepsis, pulmonary embolism, respiratory insufficiency and cardiac arrest. The clinical course of GBS complicated by acute rhabdomyolysis is extremely rare. We present the case of GBS with marked elevation in serum creatine kinase, serum myoglobin levels and persistent hyperkalemia as a result of associated acute rhabdomyolysis. Medknow Publications & Media Pvt Ltd 2014-04 /pmc/articles/PMC4033859/ /pubmed/24872655 http://dx.doi.org/10.4103/0972-5229.130577 Text en Copyright: © Indian Journal of Critical Care Medicine http://creativecommons.org/licenses/by-nc-sa/3.0 This is an open-access article distributed under the terms of the Creative Commons Attribution-Noncommercial-Share Alike 3.0 Unported, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Saxena, Amrish
Singh, Vineeta
Verma, Nitin
Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title_full Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title_fullStr Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title_full_unstemmed Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title_short Guillain-Barre syndrome complicated by acute fatal rhabdomyolysis
title_sort guillain-barre syndrome complicated by acute fatal rhabdomyolysis
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4033859/
https://www.ncbi.nlm.nih.gov/pubmed/24872655
http://dx.doi.org/10.4103/0972-5229.130577
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