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Molecular Dissection of Cyclosporin A’s Neuroprotective Effect Reveals Potential Therapeutics for Ischemic Brain Injury

After the onset of brain ischemia, a series of events leads ultimately to the death of neurons. Many molecules can be pharmacologically targeted to protect neurons during these events, which include glutamate release, glutamate receptor activation, excitotoxicity, Ca(2+) influx into cells, mitochond...

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Detalles Bibliográficos
Autor principal: Kawakami, Minoru
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2013
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4061870/
https://www.ncbi.nlm.nih.gov/pubmed/24961531
http://dx.doi.org/10.3390/brainsci3031325

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