Cargando…
Protein degradation and protein synthesis in long-term memory formation
Long-term memory (LTM) formation requires transient changes in the activity of intracellular signaling cascades that are thought to regulate new gene transcription and de novo protein synthesis in the brain. Consistent with this, protein synthesis inhibitors impair LTM for a variety of behavioral ta...
Autores principales: | Jarome, Timothy J., Helmstetter, Fred J. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Frontiers Media S.A.
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4072070/ https://www.ncbi.nlm.nih.gov/pubmed/25018696 http://dx.doi.org/10.3389/fnmol.2014.00061 |
Ejemplares similares
-
Memory formation for trace fear conditioning requires ubiquitin-proteasome mediated protein degradation in the prefrontal cortex
por: Reis, David S., et al.
Publicado: (2013) -
CaMKII, but not protein kinase A, regulates Rpt6 phosphorylation and proteasome activity during the formation of long-term memories
por: Jarome, Timothy J., et al.
Publicado: (2013) -
Activity Dependent Protein Degradation Is Critical for the Formation and Stability of Fear Memory in the Amygdala
por: Jarome, Timothy J., et al.
Publicado: (2011) -
The ubiquitin-specific protease 14 (USP14) is a critical regulator of long-term memory formation
por: Jarome, Timothy J., et al.
Publicado: (2014) -
Proteomic Analysis Reveals Sex-Specific Protein Degradation Targets in the Amygdala During Fear Memory Formation
por: Farrell, Kayla, et al.
Publicado: (2021)