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Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent

Detalles Bibliográficos
Autor principal: Ouellette, Y
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2006
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4092470/
http://dx.doi.org/10.1186/cc4442
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author Ouellette, Y
author_facet Ouellette, Y
author_sort Ouellette, Y
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spelling pubmed-40924702014-09-23 Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent Ouellette, Y Crit Care Poster Presentation BioMed Central 2006 2006-03-21 /pmc/articles/PMC4092470/ http://dx.doi.org/10.1186/cc4442 Text en Copyright © 2006 BioMed Central Ltd
spellingShingle Poster Presentation
Ouellette, Y
Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title_full Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title_fullStr Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title_full_unstemmed Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title_short Microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
title_sort microcirculatory hyporesponsiveness in lipopolysaccharide-induced inflammation is endothelial cell dependent but calcium independent
topic Poster Presentation
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4092470/
http://dx.doi.org/10.1186/cc4442
work_keys_str_mv AT ouellettey microcirculatoryhyporesponsivenessinlipopolysaccharideinducedinflammationisendothelialcelldependentbutcalciumindependent