Cargando…

Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions

Detalles Bibliográficos
Autores principales: Bowles, Neil E, Arrington, Cammon B, Hirono, Keiichi, Nakamura, Tsuneyuki, Ngo, Long, Wee, Yin Shen, Ichida, Fukiko, Weis, John H
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BlackWell Publishing Ltd 2014
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4113277/
https://www.ncbi.nlm.nih.gov/pubmed/25077179
http://dx.doi.org/10.1002/mgg3.79
_version_ 1782328269632176128
author Bowles, Neil E
Arrington, Cammon B
Hirono, Keiichi
Nakamura, Tsuneyuki
Ngo, Long
Wee, Yin Shen
Ichida, Fukiko
Weis, John H
author_facet Bowles, Neil E
Arrington, Cammon B
Hirono, Keiichi
Nakamura, Tsuneyuki
Ngo, Long
Wee, Yin Shen
Ichida, Fukiko
Weis, John H
author_sort Bowles, Neil E
collection PubMed
description
format Online
Article
Text
id pubmed-4113277
institution National Center for Biotechnology Information
language English
publishDate 2014
publisher BlackWell Publishing Ltd
record_format MEDLINE/PubMed
spelling pubmed-41132772014-07-30 Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions Bowles, Neil E Arrington, Cammon B Hirono, Keiichi Nakamura, Tsuneyuki Ngo, Long Wee, Yin Shen Ichida, Fukiko Weis, John H Mol Genet Genomic Med Letter to the Editor BlackWell Publishing Ltd 2014-07 2014-05-14 /pmc/articles/PMC4113277/ /pubmed/25077179 http://dx.doi.org/10.1002/mgg3.79 Text en © 2014 The Authors. Molecular Genetics & Genomic Medicine published by Wiley Periodicals, Inc. http://creativecommons.org/licenses/by/3.0/ This is an open access article under the terms of the Creative Commons Attribution License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.
spellingShingle Letter to the Editor
Bowles, Neil E
Arrington, Cammon B
Hirono, Keiichi
Nakamura, Tsuneyuki
Ngo, Long
Wee, Yin Shen
Ichida, Fukiko
Weis, John H
Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title_full Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title_fullStr Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title_full_unstemmed Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title_short Kawasaki disease patients homozygous for the rs12252-C variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
title_sort kawasaki disease patients homozygous for the rs12252-c variant of interferon-induced transmembrane protein-3 are significantly more likely to develop coronary artery lesions
topic Letter to the Editor
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4113277/
https://www.ncbi.nlm.nih.gov/pubmed/25077179
http://dx.doi.org/10.1002/mgg3.79
work_keys_str_mv AT bowlesneile kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT arringtoncammonb kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT hironokeiichi kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT nakamuratsuneyuki kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT ngolong kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT weeyinshen kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT ichidafukiko kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions
AT weisjohnh kawasakidiseasepatientshomozygousforthers12252cvariantofinterferoninducedtransmembraneprotein3aresignificantlymorelikelytodevelopcoronaryarterylesions