Cargando…
Hypoxia signaling controls postnatal changes in cardiac mitochondrial morphology and function
Fetal cardiomyocyte adaptation to low levels of oxygen in utero is incompletely understood, and is of interest as hypoxia tolerance is lost after birth, leading to vulnerability of adult cardiomyocytes. It is known that cardiac mitochondrial morphology, number and function change significantly follo...
Autores principales: | Neary, Marianne T., Ng, Keat-Eng, Ludtmann, Marthe H.R., Hall, Andrew R., Piotrowska, Izabela, Ong, Sang-Bing, Hausenloy, Derek J., Mohun, Timothy J., Abramov, Andrey Y., Breckenridge, Ross A. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Academic Press
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4121533/ https://www.ncbi.nlm.nih.gov/pubmed/24984146 http://dx.doi.org/10.1016/j.yjmcc.2014.06.013 |
Ejemplares similares
-
A mouse model to study the link between hypoxia, long QT interval and sudden infant death syndrome
por: Neary, Marianne T., et al.
Publicado: (2013) -
Hypoxia at the heart of sudden infant death syndrome?
por: Neary, Marianne T., et al.
Publicado: (2013) -
PKA Phosphorylation of NCLX Reverses Mitochondrial Calcium Overload
and Depolarization, Promoting Survival of PINK1-Deficient Dopaminergic
Neurons
por: Kostic, Marko, et al.
Publicado: (2015) -
Hypoxic Regulation of Hand1 Controls the Fetal-Neonatal Switch in Cardiac Metabolism
por: Breckenridge, Ross A., et al.
Publicado: (2013) -
Correction: Hypoxic Regulation of Hand1 Controls the Fetal-Neonatal Switch in Cardiac Metabolism
por: Breckenridge, Ross A., et al.
Publicado: (2013)