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The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review

Hereditary factors are increasingly attracting the interest of behavioral scientists and practitioners. Our aim in the present article is to introduce some state-of-the-art topics in behavioral genetics, as well as selected findings in the field, in order to illustrate how genetic makeup can modulat...

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Detalles Bibliográficos
Autores principales: Buades-Rotger, Macià, Gallardo-Pujol, David
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Dove Medical Press 2014
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4124068/
https://www.ncbi.nlm.nih.gov/pubmed/25114607
http://dx.doi.org/10.2147/PRBM.S40458
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author Buades-Rotger, Macià
Gallardo-Pujol, David
author_facet Buades-Rotger, Macià
Gallardo-Pujol, David
author_sort Buades-Rotger, Macià
collection PubMed
description Hereditary factors are increasingly attracting the interest of behavioral scientists and practitioners. Our aim in the present article is to introduce some state-of-the-art topics in behavioral genetics, as well as selected findings in the field, in order to illustrate how genetic makeup can modulate the impact of environmental factors. We focus on the most-studied polymorphism to date for antisocial responses to adversity: the monoamine oxidase A gene. Advances, caveats, and promises of current research are reviewed. We also discuss implications for the use of genetic information in applied settings.
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spelling pubmed-41240682014-08-11 The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review Buades-Rotger, Macià Gallardo-Pujol, David Psychol Res Behav Manag Review Hereditary factors are increasingly attracting the interest of behavioral scientists and practitioners. Our aim in the present article is to introduce some state-of-the-art topics in behavioral genetics, as well as selected findings in the field, in order to illustrate how genetic makeup can modulate the impact of environmental factors. We focus on the most-studied polymorphism to date for antisocial responses to adversity: the monoamine oxidase A gene. Advances, caveats, and promises of current research are reviewed. We also discuss implications for the use of genetic information in applied settings. Dove Medical Press 2014-07-30 /pmc/articles/PMC4124068/ /pubmed/25114607 http://dx.doi.org/10.2147/PRBM.S40458 Text en © 2014 Buades-Rotger and Gallardo-Pujol. This work is published by Dove Medical Press Limited, and licensed under Creative Commons Attribution – Non Commercial (unported, v3.0) License The full terms of the License are available at http://creativecommons.org/licenses/by-nc/3.0/. Non-commercial uses of the work are permitted without any further permission from Dove Medical Press Limited, provided the work is properly attributed.
spellingShingle Review
Buades-Rotger, Macià
Gallardo-Pujol, David
The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title_full The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title_fullStr The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title_full_unstemmed The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title_short The role of the monoamine oxidase A gene in moderating the response to adversity and associated antisocial behavior: a review
title_sort role of the monoamine oxidase a gene in moderating the response to adversity and associated antisocial behavior: a review
topic Review
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4124068/
https://www.ncbi.nlm.nih.gov/pubmed/25114607
http://dx.doi.org/10.2147/PRBM.S40458
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