Overexpression of c-Met increases the tumor invasion of human prostate LNCaP cancer cells in vitro and in vivo

c-Met is a transmembrane tyrosine kinase receptor that may be activated by hepatocyte growth factor, an inducer of epithelial-mesenchymal transition (EMT), to regulate the associated downstream gene expression. This process is critical to cell migration in normal and pathological conditions. In the...

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Detalles Bibliográficos
Autores principales: HAN, YILI, LUO, YONG, ZHAO, JIAHUI, LI, MINGCHUAN, JIANG, YONGGUANG
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2014
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4156182/
https://www.ncbi.nlm.nih.gov/pubmed/25202379
http://dx.doi.org/10.3892/ol.2014.2390

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4156182/
https://www.ncbi.nlm.nih.gov/pubmed/25202379
http://dx.doi.org/10.3892/ol.2014.2390

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