Cargando…
Angiotensin II Removes Kidney Resistance Conferred by Ischemic Preconditioning
Ischemic preconditioning (IPC) by ischemia/reperfusion (I/R) renders resistance to the kidney. Strong IPC triggers kidney fibrosis, which is involved in angiotensin II (AngII) and its type 1 receptor (AT1R) signaling. Here, we investigated the role of AngII/AT1R signal pathway in the resistance of I...
Autores principales: | Jang, Hee-Seong, Kim, Jee In, Kim, Jinu, Park, Jeen-Woo, Park, Kwon Moo |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Hindawi Publishing Corporation
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4163347/ https://www.ncbi.nlm.nih.gov/pubmed/25243156 http://dx.doi.org/10.1155/2014/602149 |
Ejemplares similares
-
Intra-renal slow cell-cycle cells contribute to the restoration of kidney tubules injured by ischemia/reperfusion
por: Kim, Jinu, et al.
Publicado: (2011) -
Mitochondrial NADP(+)-dependent isocitrate dehydrogenase deficiency increases cisplatin-induced oxidative damage in the kidney tubule cells
por: Kong, Min Jung, et al.
Publicado: (2018) -
Fragmentation of kidney epithelial cell primary cilia occurs by cisplatin and these cilia fragments are excreted into the urine
por: Kong, Min Jung, et al.
Publicado: (2018) -
Unilateral nephrectomy elongates primary cilia in the remaining kidney via reactive oxygen species
por: Han, Sang Jun, et al.
Publicado: (2016) -
Role of Remote Ischemic Preconditioning in Hepatic Ischemic Reperfusion Injury
por: Choi, Eun Kyung, et al.
Publicado: (2020)