Cargando…
An Intracellular Ca(2+) Channel is Required For Sarcolemma Repair to Prevent Muscular Dystrophy
The integrity of the plasma membrane is maintained through an active repair process, especially for skeletal and cardiac muscle cells, in which contraction-induced mechanical damage frequently occurs in vivo(1,2). Muscular dystrophies (MDs) are a group of muscle diseases characterized by skeletal mu...
Autores principales: | Cheng, Xiping, Zhang, Xiaoli, Gao, Qiong, Samie, Mohammad Ali, Azar, Marlene, Tsang, Wai Lok, Dong, Libing, Sahoo, Nirakar, Li, Xinran, Zhuo, Yue, Garrity, Abigail G., Wang, Xiang, Ferrer, Marc, Dowling, James, Xu, Li, Han, Renzhi, Xu, Haoxing |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4192061/ https://www.ncbi.nlm.nih.gov/pubmed/25216637 http://dx.doi.org/10.1038/nm.3611 |
Ejemplares similares
-
Myogenic Akt signaling upregulates the utrophin–glycoprotein complex and promotes sarcolemma stability in muscular dystrophy
por: Peter, Angela K., et al.
Publicado: (2009) -
Small-molecule activation of lysosomal TRP channels ameliorates Duchenne muscular dystrophy in mouse models
por: Yu, Lu, et al.
Publicado: (2020) -
Metformin Increases Sarcolemma Integrity and Ameliorates Neuromuscular Deficits in a Murine Model of Duchenne Muscular Dystrophy
por: Dong, Xia, et al.
Publicado: (2021) -
A voltage-dependent K(+) channel in the lysosome is required for refilling lysosomal Ca(2+) stores
por: Wang, Wuyang, et al.
Publicado: (2017) -
Human Galectin-1 Improves Sarcolemma Stability and Muscle Vascularization in the mdx Mouse Model of Duchenne Muscular Dystrophy
por: Wuebbles, Ryan D., et al.
Publicado: (2019)