Cargando…
Arginase 2 Deficiency Prevents Oxidative Stress and Limits Hyperoxia-Induced Retinal Vascular Degeneration
BACKGROUND: Hyperoxia exposure of premature infants causes obliteration of the immature retinal microvessels, leading to a condition of proliferative vitreoretinal neovascularization termed retinopathy of prematurity (ROP). Previous work has demonstrated that the hyperoxia-induced vascular injury is...
Autores principales: | Suwanpradid, Jutamas, Rojas, Modesto, Behzadian, M. Ali, Caldwell, R. William, Caldwell, Ruth B. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4222858/ https://www.ncbi.nlm.nih.gov/pubmed/25375125 http://dx.doi.org/10.1371/journal.pone.0110604 |
Ejemplares similares
-
Arginase 2 Deletion Reduces Neuro-Glial Injury and Improves Retinal Function in a Model of Retinopathy of Prematurity
por: Narayanan, Subhadra P., et al.
Publicado: (2011) -
Arginase 2 deficiency reduces hyperoxia-mediated retinal neurodegeneration through the regulation of polyamine metabolism
por: Narayanan, S P, et al.
Publicado: (2014) -
Targeting proliferative retinopathy: Arginase 1 limits vitreoretinal neovascularization and promotes angiogenic repair
por: Fouda, Abdelrahman Y., et al.
Publicado: (2022) -
Arginase 2 promotes neurovascular degeneration during ischemia/reperfusion injury
por: Shosha, Esraa, et al.
Publicado: (2016) -
NOX2-Induced Activation of Arginase and Diabetes-Induced Retinal Endothelial Cell Senescence
por: Rojas, Modesto, et al.
Publicado: (2017)