Cargando…
IL-17A and Serum Amyloid A Are Elevated in a Cigarette Smoke Cessation Model Associated with the Persistence of Pigmented Macrophages, Neutrophils and Activated NK Cells
While global success in cessation advocacy has seen smoking rates fall in many developed countries, persistent lung inflammation in ex-smokers is an increasingly important clinical problem whose mechanistic basis remains poorly understood. In this study, candidate effector mechanisms were assessed i...
Autores principales: | Hansen, Michelle J., Chan, Sheau Pyng J., Langenbach, Shenna Y., Dousha, Lovisa F., Jones, Jessica E., Yatmaz, Selcuk, Seow, Huei Jiunn, Vlahos, Ross, Anderson, Gary P., Bozinovski, Steven |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2014
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4236152/ https://www.ncbi.nlm.nih.gov/pubmed/25405776 http://dx.doi.org/10.1371/journal.pone.0113180 |
Ejemplares similares
-
Innate cellular sources of interleukin-17A regulate macrophage accumulation in cigarette- smoke-induced lung inflammation in mice
por: Bozinovski, Steven, et al.
Publicado: (2015) -
Ebselen reduces cigarette smoke‐induced endothelial dysfunction in mice
por: Brassington, Kurt, et al.
Publicado: (2021) -
Tumour-associated neutrophils and loss of epithelial PTEN can promote corticosteroid-insensitive MMP-9 expression in the chronically inflamed lung microenvironment
por: Vannitamby, Amanda, et al.
Publicado: (2017) -
Glutathione Peroxidase-1 Primes Pro-Inflammatory Cytokine Production after LPS Challenge In Vivo
por: Bozinovski, Steven, et al.
Publicado: (2012) -
Influenza A virus infection and cigarette smoke impair bronchodilator responsiveness to β-adrenoceptor agonists in mouse lung
por: Donovan, Chantal, et al.
Publicado: (2016)