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Our “energy-Ca(2+) signaling deficits” hypothesis and its explanatory potential for key features of Alzheimer’s disease
Sporadic Alzheimer’s disease (sAD) has not been explained by any current theories, so new hypotheses are urgently needed. We proposed that “energy and Ca(2+) signaling deficits” are perhaps the earliest modifiable defects in brain aging underlying memory decline and tau deposits (by means of inactiv...
| Autores principales: | , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
Frontiers Media S.A.
2014
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4253736/ https://www.ncbi.nlm.nih.gov/pubmed/25489296 http://dx.doi.org/10.3389/fnagi.2014.00329 |