Cargando…
Kaposi's sarcoma-associated herpes virus disrupts adherens junctions and increases endothelial permeability by inducing degradation of VE-cadherin
Autores principales: | Qian, LW, Greene, W, Ye, FC, Gao, SJ |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2009
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4261772/ http://dx.doi.org/10.1186/1750-9378-4-S2-P18 |
Ejemplares similares
-
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
por: Tiruppathi, Chinnaswamy, et al.
Publicado: (2023) -
CMTM4 regulates angiogenesis by promoting cell surface recycling of VE-cadherin to endothelial adherens junctions
por: Chrifi, Ihsan, et al.
Publicado: (2018) -
N-cadherin signaling via Trio assembles adherens junctions to restrict endothelial permeability
por: Kruse, Kevin, et al.
Publicado: (2019) -
Fibroblast Growth Factor Signaling Potentiates VE-Cadherin Stability at Adherens Junctions by Regulating SHP2
por: Hatanaka, Kunihiko, et al.
Publicado: (2012) -
Evidence of a common mechanism of disassembly of adherens junctions through Gα13 targeting of VE-cadherin
por: Gong, Haixia, et al.
Publicado: (2014)