Altering endoplasmic reticulum stress in a model of blast-induced traumatic brain injury controls cellular fate and ameliorates neuropsychiatric symptoms

Neuronal injury following blast-induced traumatic brain injury (bTBI) increases the risk for neuropsychiatric disorders, yet the pathophysiology remains poorly understood. Blood-brain-barrier (BBB) disruption, endoplasmic reticulum (ER) stress, and apoptosis have all been implicated in bTBI. Microve...

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Detalles Bibliográficos
Autores principales: Logsdon, Aric Flint, Turner, Ryan Coddington, Lucke-Wold, Brandon Peter, Robson, Matthew James, Naser, Zachary James, Smith, Kelly Elizabeth, Matsumoto, Rae Reiko, Huber, Jason Delwyn, Rosen, Charles Lee
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2014
Materias:
Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4261829/
https://www.ncbi.nlm.nih.gov/pubmed/25540611
http://dx.doi.org/10.3389/fncel.2014.00421

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4261829/
https://www.ncbi.nlm.nih.gov/pubmed/25540611
http://dx.doi.org/10.3389/fncel.2014.00421

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