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Connexin26 hemichannels with a mutation that causes KID syndrome in humans lack sensitivity to CO(2)
Mutations in connexin26 (Cx26) underlie a range of serious human pathologies. Previously we have shown that Cx26 hemichannels are directly opened by CO(2) (Meigh et al., 2013). However the effects of human disease-causing mutations on the CO(2) sensitivity of Cx26 are entirely unknown. Here, we repo...
| Autores principales: | , , , |
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| Formato: | Online Artículo Texto |
| Lenguaje: | English |
| Publicado: |
eLife Sciences Publications, Ltd
2014
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4270064/ https://www.ncbi.nlm.nih.gov/pubmed/25422938 http://dx.doi.org/10.7554/eLife.04249 |